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ELIZABETH M. HAYNES, PHD
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Elizabeth "Liz" Haynes is a cell biologist studying neurodevelopment using zebrafish in the lab of Dr. Mary Halloran at UW-Madison. She is co-mentored in microscopy and image analysis by Dr. Kevin Eliceiri at LOCI, and collaborates with the Huisken lab to do medium-throughput CRISPR F0 screening using Light Sheet Microscopy.

She thinks of neurons as just really fancy cells.


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She received her Ph.D. in Cell Biology and Physiology from UNC Chapel Hill, where she studied actin debranching under the mentorship of Dr. Jim Bear.

She received her B.S. in Molecular Biology and Microbiology from UCF, with an Honor's Thesis on immunotoxin-based cancer therapies in the lab of Dr. Kenneth Teter.

Fellowships: 
  • NIH F32 (F32NS098689-03)
  • Micheal Guyer Post-Doctoral Fellowship
  • American Heart Association Pre-Doctoral Fellowship
  • Department of Defense NCMR Research Scholarship
  • American Cancer Society Undergraduate Research Fellowship

Select Publications
  • The Kinesin Adaptor Calsyntenin-1 Organizes Microtubule Polarity and Regulates Dynamics during Sensory Axon Arbor Development. Lee TJ, Lee JW, Haynes EM, Eliceiri KW, Halloran MC. Frontiers in Neuroscience. 2017.
  • Lamellipodia are crucial for haptotactic sensing and response. King SJ, Asokan SB, Haynes EM, Zimmerman SP, Rotty JD, Alb JG. Jr., Blake D, Tagliatela A, Lebedeva IP, Marston D, Johnson HE, Parsons M, Sharpless NE, Kuhlman B, Haugh JM, Bear JE. J. Cell. Sci. 2016.
  • Profilin-1 serves as a gatekeeper for actin assembly by Arp2/3-dependent and -independent pathways. Rotty JD, Wu C, Haynes EM, Suarez C, Winkelman JD, Johnson HE, Haugh JM, Kovar DR, Bear JE. Dev. Cell. 2015.
  • GMFβ controls branched actin content and lamellipodial retraction in fibroblasts. Haynes EM, Asokan SB, King SJ, Johnson HE, Haugh JM, Bear JE. JCB. 2015.
  • Loss of Arp2/3 induces an NF-κB-dependent, nonautonomous effect on chemotactic signaling. Wu C, Haynes EM, Asokan SB, Simon JM, Sharpless NE, Baldwin AS, Davis IJ, Johnson GL, Bear JE. JCB. 2013.
  • Arp2/3 is critical for lamellipodia and response to extracellular matrix cues but is dispensable for chemotaxis. Wu C, Asokan SB, Berginski ME, Haynes EM, Sharpless NE, Griffith JD, Gomez SM, and Bear JE. Cell. 2012. 



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